PostHeaderIcon Acute Reticulosis

(Internal Disease - Circulatory)

blood cells

Acute reticulosis - a form of acute leukemia characterized by a hyperplastic process involving the reticular tissue of the stroma-forming organs and by aleukemic blood picture, in which the undifferentiated and immature hematopoietic elements do not enter the peripheral blood, or get it in small quantities.

Symptoms and flow. Is rare and occurs usually in the absence of clinical symptoms of hyperplasia of hematopoietic organs, but with rapidly progressive pancytopenia. It is characterized by sudden fever appears to 39-40, severe intoxication, sharp pains in the bones and joints.

According to the clinical picture and the composition of peripheral blood resembles apl [more..]

PostHeaderIcon Hemophilia

(Internal Disease - Circulatory)

blood cells

Hemophilia - a form of hemorrhagic diathesis, in which the tendency to bleeding due to an impaired blood coagulation due to deficiency of plasmic thromboplastin generation.

Etiology and pathogenesis. Hemophilia occurs in the form of congenital, rarely sporadic forms. Hemophiliacs suffer from male carriers of the disease are women.

A-form of Hemophilia associated with the lack anti-hemophilic globulin and occurs in 80-90% of patients with hemophilia.

B-form of Hemophilia is caused by lack of the plasmic thromboplastin component, occurs in 10-15% of patients with hemophilia.

C-form of Hemophilia characterized by lack of plasmic thromboplastin p [more..]

PostHeaderIcon Acquired Hemolytic Anemia

(Internal Disease - Circulatory)

blood cells

Acute Acquired Hemolytic Anemia

Etiology and pathogenesis. Arise as a result of infections (sepsis, malaria, etc.), intoxications hemolytic poisons (phosphorus, phenylhydrazine, or mushroom, snake venom, etc.), physical factors (cooling, burns). Increased hemolysis arises from the direct effects on red blood cells of the pathogenic factor and as a result of the influence of exogenous factors that lead to the formation of anti-erythrocyte antibodies in the application of certain medicinal substances. Thus, the use of Amidopyrine, Quinine, sulfanilamide and other drugs can lead to the formation of autoantibodies. As a result, connecting them with proteins and blood cells, t [more..]

PostHeaderIcon Hypotension

(Internal Disease - Cardiovascular)

heart electrical circuit

When the primary decrease in arterial hypotension and venous pressure is determined dysfunction neurohumoral system, regulating vascular tone. Secondary hypotension observed as a symptom with various infectious diseases and intoxications, ulcers and Addison’s disease.

Symptoms and flow. Complaints may be absent. Frequently observed functional disorders of the nervous and cardiovascular systems, as manifested by headaches, dizziness, general weakness, irritability, there are unpleasant sensations and pain in the heart, palpitations, sweating. Arterial and venous pressure is lowered (blood pressure 100/50 - 80/40 mm Hg. Art., Venous pressure below 80-60 [more..]

PostHeaderIcon Congenital Hemolytic Anemia

(Internal Disease - Circulatory)

blood cells

Thalassemia

Etiology and pathogenesis. The disease is inherited family trait the predominance of red blood cells of fetal embryonic hemoglobin.

Symptoms and flow. There are large, small and minimal thalassemia. The first form of thalassemia in children-homozygotes, the latter two forms a child of heterozygotes. A typical symptom for thalassemia major is a progressive anemia with erythroblastosis, enlarged spleen and liver, osteoporosis, with peculiar changes in bones of the skull (disproportionate proliferation of parietal and occiput) as a consequence of abnormal proliferation of erythroblastic germ in the bone marrow. In the blood, anemia with eritroblasto [more..]

PostHeaderIcon Tendosynovitis

(Internal Disease - Joints tissue)

joints and periarticular tissue

Tendosynovitis - diseases of tendons and tendon sheaths. Generally affecting the extensor tendons of the muscles of hand and foot going through the narrow bone-fibrous feeds. As a consequence, the tendons exposed to minor trauma that leads to the development of the synovial membrane of a small tendon inflammation, which later can turn into chronic and lead to compaction and stenosis of the tendon.

Tendosynovitis of the hands develops mainly in women engaged in manual labor (dressmakers, typists, pianists, etc.). Is usually found in the long adductor tendon and the short extensor muscle of thumb, which manifests itself with pain in the area su [more..]

PostHeaderIcon Endemic Deforming Arthritis

(Internal Disease - Joints)

joints and periarticular tissue

Endemic deforming arthritis - generalized osteoarthritis deformans with profound violation of the ossification process. Origin is unknown. It is believed that matter eating cereals contaminated by the fungus Fusarium sporotrichiella, or lack of calcium in water and soil. Well, they get children 6-14 years old. In the epiphysis of tubular bones develops a deep degenerative process (necrosis of cartilage, changes in articular surfaces with abundant marginal bone growths), leading to significant disfigurement of joints and bone dysplasia (short fingers, short stature).

Symptoms and flow. Characterized by slow progression of the disease with the gradual [more..]

PostHeaderIcon Nonspecific Infectious Polyarthritis

(Internal Disease - Collagen)

joints and periarticular tissue

Etiology and pathogenesis are not sufficiently clear. It is assumed that this common infectious-allergic disease associated with the presence in the body the focus of chronic infection (chronic tonsillitis, otitis, cholecystitis, etc.). Continuous flow in the blood of toxins from the infection focus leads to a change in immunological reactivity and the development of allergy. In the process of disorganization of connective tissue (mainly tissue of the joints), released a number of substances purchased properties autoantigens, against which antibodies are formed and the whole process takes autoimmune in nature, manifested by persistent, progressive course. In the blood of pat [more..]

PostHeaderIcon Hypertension

(Internal Disease - Cardiovascular)

heart electrical circuit

Etiology and pathogenesis. There is a neurosis regulating blood pressure points, leading to increased muscle tone of the arterial wall, narrowing of the lumen of small arteries and arterioles and increase blood pressure. Trigger a strain neuro-psychological sphere. For secondary mechanisms include renal ischemic and endocrine factors. Ischemia kidney juxtaglomerular apparatus allocates renin, which ultimately leads to increased blood pressure.

Increased secretion of aldosterone leading to sodium retention and accumulation of fluid in the walls of arterioles, which narrows their lumen and increases the pressure.

Symptoms and flow. Hypertensive [more..]

PostHeaderIcon Chronic Posthemorrhagic Anemia

(Internal Disease - Circulatory)

blood cells

Chronic Posthemorrhagic Anemia is also known as Thalassemia or Cooley’s Anaemia.

Etiology. Arises as a result of repeated bleeding for a long period.

Symptoms and flow is characterized by gradually developing weakness, shortness of breath with movements, heart palpitations, dizziness. Pale skin and visible mucous membranes. The picture of blood to the forefront decline in hemoglobin and red blood cells. As a result of chronic bleeding there is increased loss of body iron stores, which affects a hypochromia of erythrocytes and thus to reduce the color index of blood to 0,6-0,7. Reveals the degenerative forms of erythrocytes: poykilotsity, anizotsity. Th [more..]

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